SCAR TREATMENT

Scar Treatment After Healing from Injury

Topical application of our biological skin cream for scar treatment on skin wounds and scars regulates dermal fibroblast proliferation, and will prevent or avoid fibrosis, inhibit and decrease keloid scars and hyperthropic scars.

Scars are the pale pink and brown or silvery patches on the skin that form after the skin has experienced some sort of injury. It's the skins natural way of repairing itself.

Scars are a part of life and each one tells a story, however sometimes the scars are unsightly and embarrassing and may have negative affects in many aspects of your life.

The human body is able to withstand many different types of injuries, including penetrating trauma, burn trauma and blunt trauma. All of these insults set into motion an orderly succession of events that represent a healing response in which the skin is involved. The natural process re-establishes integrity by forming connective tissue (resulting in scars) and this response is characterized by the movement of specialized cells into the injury site.

When an injury occurs a migration of diverse cells begins immediately in the area and the complex healing process starts the moment an injury or wound happens.

Platelets and inflammatory cells are the first cells to appear at the site of injury and they generate key "signals" needed for the influx of connective tissue cells and a new blood supply. This phase is called inflammatory phase. The signs have been well recognized since ancient times: rubor (redness), calor (heat), tumor (swelling) and dolor (pain).

The arrival of wound macrophages is a marker that the inflammatory phase is nearing an end and the proliferative phase will begin. Lymphocytes appear at the wound area in a latter phase but are not contemplated to be major inflammatory cells involved in the healing reaction; their precise role in the wound healing system remains unclear.

As the Proliferative phase advances the predominant cell in the wound site is the fibroblast. This cell is in charge of producing the new matrix necessary to restitute structural integrity and function to the injured tissue. Fibroblasts attach to the cables of the provisional fibrin matrix and begin to generate collagen.

Collagen is the most abundant protein in the animal kingdom, accounting for 30% of the entire protein in the human body. In normal tissues collagen gives strength, stability and structure. Collagen is necessary to repair the defect and regenerate anatomic structures and function. If too much collagen accumulates in the wound site, natural anatomical structures are deteriorated, function is compromised and fibrosis appears.

In summary, the normal healing cascade starts with a systematized process of homeostasis and fibrin deposition, which leads to an inflammatory cell cascade, distinguish by neutrophils, macrophages and lymphocytes within the tissue.

This is followed by attraction and proliferation of fibroblasts and collagen deposition, and finally remodelling by collagen cross-linking and scar formation. Although this orderly chain of events is responsible for normal wound healing, pathologic responses leading to fibrosis or chronic ulcers might take place if any part of the healing chain is altered.

Fibrosis: Abnormal Skin Reaction to Injury

Fibrosis is defined as the equivalent of the normal structural components of the tissue by alteration, non-functional and excessive accumulation of scar tissue. This is maybe the most crucial biological marker for fibrosis. A good number of clinical problems are associated with excessive scar formation for example, keloids and hypertrophic scars.

Keloid scars are basically thick, puckered, itchy clusters of scar tissue that arise beyond the borders of an injury or incision and almost never regress. Keloid scars are sometimes very nodular in nature, and they are typically darker in color than surrounding skin. They take place when the body persists in produce tough, fibrous protein (known as collagen) once a wound has healed.

Keloids are fibrotic tumors defined by a collection of abnormal fibroblasts with excessive deposition of collagen, fibronectin, elastin, and proteoglycans. Histologically, keloids contain relatively acellular centers and thick, abundant collagen bundles that create nodules in the deep dermal portion of the lesion.

Keloids present a therapeutic challenge that must be addressed while these lesions can cause substantial pain, pruritus (severe itching) and physical disfigurement. They unfortunately seldom improve in aspect over time, and can even limit mobility if the form over a joint.

Keloids can occur anywhere on the body, but frequently occur over the breastbone, on earlobes and on shoulders. Keloids mainly emerge in people with dark skin, such as individuals of Asian, African or Middle-Eastern descent. A person's tendency to develop keloids does decrease with age. But, one of the most troublesome aspects of keloids scars is their tendency to recur, occasionally needing permanent care.

Hypertrophic scars rarely are difficult to identify from keloid scars histologically and biochemically. Unlike keloids, hypertrophic scars stay confined to the wound site and normally mature and flatten out over time.

Both forms secrete higher concentrations of collagen than normal scars, but usually the hypertrophic type exhibits declining collagen synthesis after about six months. But, hypertrophic scars have nearly twice as much glycosaminoglycan as normal scars and this and enhanced synthetic and enzymatic activity end in substantial alterations in the matrix which impacts the mechanical properties of the scars, including reduced extensibility that makes them feel firm.

As with hypertrophic scarring, people who already have one keloid scar are more likely to suffer from this syndrome in the future and have to alert their doctor or surgeon if they need injections or are to have any form of surgery.

BIOSKINCARE™ Treatment cream contributes to REGULATE the skin healing, scar formation process and all associated fibrosis.

BIOSKINCARE™ is an organic cream that can help you to improve the levels of collagen, the most important elements of the skin regeneration mechanism. It is a natural cosmetic treatment cream, not a drug, pharmaceutical, or product aimed to cure a disease.

BIOSINCARE's main component is a secretion from the land snail (Helix Aspersa Muller). It is the same that they employ to quickly repair their own skin and shells when injured. It is a viscose liquid that is made into an un-perfumed white cream with no alien or synthetic chemicals. Topical application of the cream on skin wounds and scars reduces dermal fibroblast proliferation, and inhibits and decreases keloid and hyperthropic scarring.

Analysis of the secretion of the snail has shown that it contains biological activators that enhance the skin's growth factors which in turn reproduce brand new skin cells. It also contains antimicrobial peptides, and antioxidants that act as natural anti inflammatories and immune modulating glycoproteins. Their activity helps also in the production of glycosaminoglycans which are responsible for holding the water molecules in normal healthy skin. The skin is nourished with all the ingredients necessary to regenerate the elastin and collagen levels, which results in skin looking healthy again, quickly.

In simpler words; BIOSKINCARE™ cream helps your skin regenerate itself! It is additionally anti-microbial, thereby helping to avoid skin infections. There is nothing like it! And you can use it for treating a wide variety of skin blemishes: wounds, burns, sunburns, and rashes, cracked and dry skin. It works excellently on post-surgical scars. It is also ideal for wrinkles, age spots, abrasions, acne, athlete's foot, boils, chilblains, diabetic sores, diaper rash, infected nails, insect bites/stings, skin allergies, skin ulcers and stretch marks with incredible results. It is just fantastic for helping the skin to recover itself. Wounds heal with minimal scarring while it also helps shrink old scars.

Scar treatment with skin cream on skin wounds and scars to reduce dermal fibroblast proliferation, and inhibit and decrease keloid and hyperthropic scars